Lasten Borrelioosia käsittelevä sivusto http://www.childrenslymenetwork.org/
Allaolevassa artikkelissa on esitetty useita lasten borrelioosissa mahdollisesti esiintyviä oireita.
http://lymedisease.org/resources/children.html
Children
The CDC says that Lyme disease is most common among boys aged 5-19. This age group is affected at three times the average rate of all other age groups. Around 25% of all reported cases are children.
Children with Lyme disease have special issues. Since they did not have much of a history of wellness prior to becoming ill, Tthey don’t know what “normal” is. They can’t always explain what is feels wrong. Because the symptoms of Lyme disease can be non-specific, vague, and changeable, parents and teachers may suspect them of malingering or making things up to gain attention. It is also difficult for parents to discern when their child’s symptoms are worse or better, given the difficulties children have making that determination themselves.
Children with Lyme disease may miss important developmental stages, due to because of social isolation caused by chronic ill health, and the failure of their peers to understand the nature and degree of their illness. They may fall behind their peers in school because their brains are not functioning properly. Children suffer when their bodies hurt, when the illness causes them not to be able to have restorative sleep every night, when they must struggle in school, when they don’t even feel like playing. They may feel confused, lost, and betrayed by caregivers who fail to recognize that something organic is going on, but instead blame them.. Isolation from parents occurs when parents don’t understood the nature of the illness, and the implications for the child’s functioning.
According to research, children are bitten by ticks more frequently around the head and neck, making them more vulnerable to brain and central nervous system infections. The resulting neurologic symptoms of Lyme disease are often misdiagnosed. Lyme pediatric specialist Charles Ray Jones, MD, compiled a list of common symptoms of infection in his young patients:
severe fatigue unrelieved by rest
insomnia
headaches
nausea, abdominal pain
impaired concentration
poor short-term memory
inability to sustain attention
difficulty thinking and expressing thoughts
difficulty reading and writing
being overwhelmed by schoolwork
difficulty making decisions
confusion
uncharacteristic behavior
outbursts and mood swings
fevers/chills
joint pain
dizziness
noise and light sensitivity
Dr. Jones has also documented congenital, or gestational, Lyme disease in some children who were infected in utero or by breastfeeding. In these patients his suspicion is raised when the child has:
frequent fevers
increased incidence of ear and throat infections
increased incidence of pneumonia
irritability
joint and body pain
poor muscle tone
gastroesophageal reflux
small windpipe (tracheomalacia)
cataracts and other eye problems
developmental delay
learning disabilities
psychiatric problems
Among Jones’ patients, 50% have no known history of deer tick attachments and fewer than 10% have a history of an erythema migrans Lyme rash (bull’s-eye).
According to neuropsychiatrist Brian Fallon, MD, director of theSurveys University Lyme Disease Research Center and principal investigator of an NIH-funded study on chronic Lyme disease, about 15 percent of infected patients (not necessarily of children) develop objective neurologic abnormalities, most commonly displaying part of the triad of aseptic meningitis, cranial neuritis, and motor sensory radiculitis.
Case reports (again, not necessarily of children) have linked a variety of neurologic syndromes to late Lyme disease, including:
Blindness
Progressive demyelinating-like syndromes (mimicking Multiple sclerosis)
Amyotrophic lateral sclerosis (ALS)
Guillain-Barre
Progressive dementias
Seizure disorders
Strokes
Extrapyramidal disorders
Encephalomyelitis
Spastic paraparesis
Ataxia
Bladder dysfunction
Cranial neuropathy
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Daniel sairastui 15-vuotiaana voimakkaaseen fatiikkiin ja kävelyvaikeuksiin yms. Kahdeksan vuoden sairastelun ja epätietoisuuden jälkeen hänellä diagnosoitiin borrelioosi. Lokakuussa 2007 Daniel osallistui borrelioosia sairastavien lasten tapaamiseen, jonka tarkoituksena on herättää huomiota borrelioosiin. Borrelioosi aiheuttaa lapsille usein esim. kognitiivisia häiriöitä, fatiikkia, muistinmenetystä, sydänsairauksia, masennusta jne. Joidenkin mielestä kuukauden antibioottihoito on riittävä, toisten mielestä taudin hoito sen sijaan edellyttää pitkää antibiottihoitoa. Lapset sairastuvat tautiin useimmiten 5 - 14 ikävuoden välillä. Nykyiset borrelioositestit jättävät havaitsematta jopa 75 %:ssa tautitapauksista.
(Suom.huom. Nykyisillä testeillä (2009) ei kyetä varmuudella osoittamaan sairastaako henkilö antibioottihoidon saatuaan edelleen borrelioosia vai ei.) Families affected by Lyme disease attend forum
BY KENNY PORPORA |
lidesk@newsday.com October 21, 2007
Karen Hassan sat beside her bedridden son, unsure of what was happening. Daniel, 15 at the time, was suffering from seizures and extreme fatigue, often unable to move. She thought he was going to die, she said, and spent every night beside him so he wouldn't be alone.
After a decade of confusion, Daniel Hassan of Brookhaven, now 23, was diagnosed last year with Lyme disease.
He, along with six others fighting the disease, read an account of his experience yesterday before those attending a "Children of Lyme" town-hall-style meeting at the Manorville Fire Department.
The meeting was a platform to raise awareness about Lyme disease and discuss the struggle against it. The best treatment for Lyme is a matter of debate, with some advocating long-term medicating of patients and others saying that can be harmful.
Eva Haughie, president of the Empire State Lyme Disease Association, the sponsor of yesterday's meeting, said her group and others, including the International Lyme and Associated Disease Society, believe long-term medication is needed. They cite patients whose symptoms recurred for up to 10 months after treatment began.
On the other side of the debate, the Infectious Disease Society of America and some physicians say Lyme disease can be cured in 28 days with antibiotics. Longer treatment, the society says, could cause serious illness or allow patients to develop a tolerance for the antibiotic, rendering it ineffective for future use.
Lyme disease is a potentially life-threatening, tick-borne infection. According to the national Centers for Disease Control and Prevention, in 2005 there were more than 233,000 cases reported nationwide, 55,650 of which were New York residents. The disease most often affects children ages 5-14, according to the CDC report.
Commonly associated with causing cognitive dysfunction, fatigue, memory loss and stunted development, Lyme disease also can result in heart disease and depression, said Diane Blanchard, co-president of Time for Lyme Inc., a nonprofit organization based in Greenwich, Conn., which endorses long-term medication for patients.
Blanchard cited an October 2005 study by Johns Hopkins University Medical Center that showed current Lyme tests miss 75 percent of cases, saying more sophisticated diagnostic regimens are needed.
Since he was 12, Daniel Hassan told the group, he has been hospitalized five times in three Long Island hospitals. In August 2006, he underwent a spinal tap, when a doctor confirmed he had Lyme.
Listening in the audience was Jane Mills, 44, of West Hartford, Conn. Her son Stephen, 9, was bitten by a deer tick when he was five and diagnosed with Lyme disease. Subsequently, his knees swelled from babesiosis and bartonella, two infections that frequently accompany Lyme, and he spent his first-grade year in a wheelchair.
"He missed 37 days of school last year," his mother said. "One year he's at the top of his class, the next he needs to be placed in a special learning program."
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Borreliabakteeri aiheuttaa toisinaan ihon morpheaa lapsille ja aikuisille. "Paikallinen, ihoon rajoittuva muoto (toiselta nimeltään morphea) on tavallisin lapsilla ja nuorilla. Siinä ihoon ilmaantuu vaaleanpunervia, kiinteän arpimaisia läiskiä tai juosteita, jotka pehmenevät ja ohenevat pikkuhiljaa, kuukausien mittaan. Tämä skleroderman muoto ei yleensä vaikuta haitallisesti muualle elimistöön ja myöhemmin ei uusia muutoksia enää ilmaannu.http://www.harvinaiset.fi/diagnoosit/s4.html.
J Am Acad Dermatol. 2008 Nov 18; [Epub ahead of print]
"Borrelia-associated early-onset morphea": A particular type of scleroderma inchildhood and adolescence with high titer antinuclear antibodies? Results of acohort analysis and presentation of three cases. Prinz JC, Kutasi Z, Weisenseel P, Poto L, Battyani Z, Ruzicka T. Department of Dermatology, University of Munich, Germany.
BACKGROUND: Morphea is an inflammatory autoimmune skin sclerosis of unknownetiology. A causative role of Borrelia burgdorferi infection has beencontroversially discussed, but no conclusive solution has yet been achieved. OBJECTIVE: Intrigued by 3 young patients with severe Borrelia-associated morpheaand high-titer antinuclear antibodies, we retrospectively examined therelationship between Borrelia exposure, serologic autoimmune phenomena and ageat disease onset in morphea patients. METHODS: In 90 morphea patients thepresence of Borrelia-specific serum antibodies was correlated to the age atdisease onset and the presence and titers of antinuclear antibodies. Patientswith active Borrelia infection or high-titer antinuclear antibodies due tosystemic sclerosis or lupus erythematosus served as controls. RESULTS: We observed a statistically highly significant association between morphea, serologic evidence of Borrelia infection, and high-titer antinuclear antibodies when disease onset was in childhood or adolescence. LIMITATIONS: Because pathogenic Borrelia species may vary in different geographic regions the relevance of Borrelia infection in morphea induction may show regional variations. CONCLUSION: B burgdorferi infection may be relevant for theinduction of a distinct autoimmune type of scleroderma; it may be called"Borrelia-associated early onset morphea" and is characterized by thecombination of disease onset at younger age, infection with B burgdorferi, andevident autoimmune phenomena as reflected by high-titer antinuclear antibodies.As exemplified by the case reports, it may take a particularly severe course andrequire treatment of both infection and skin inflammation. PMID: 19022534 [PubMed - as supplied by publisher]
Kirjoittaja soijuv » Ke Helmi 04, 2009 15:27